Inhibition of fatty acid oxidation promotes macrophage control of Mycobacterium tuberculosis
Fatty acid oxidation of alternatively activated macrophages prevents foam cell formation, but Mycobacterium tuberculosis counteracts this process via HIF-1α activation | PLOS Pathogens
PDF] Etomoxir Actions on Regulatory and Memory T Cells Are Independent of Cpt1a-Mediated Fatty Acid Oxidation. | Semantic Scholar
Fatty acid oxidation of alternatively activated macrophages prevents foam cell formation, but Mycobacterium tuberculosis counteracts this process via HIF-1α activation | PLOS Pathogens
Etomoxir Inhibits Macrophage Polarization by Disrupting CoA Homeostasis - ScienceDirect
Cancers | Free Full-Text | Impact of Lipid Metabolism on Antitumor Immune Response
Etomoxir Inhibits Macrophage Polarization by Disrupting CoA Homeostasis - ScienceDirect
Fatty acid oxidation in macrophage polarization. - Abstract - Europe PMC
Etomoxir Inhibits Macrophage Polarization by Disrupting CoA Homeostasis - ScienceDirect
Fa(c)t checking: How fatty acids shape metabolism and function of macrophages and dendritic cells - Almeida - 2021 - European Journal of Immunology - Wiley Online Library
Mitohormesis reprograms macrophage metabolism to enforce tolerance | bioRxiv
Lipid droplet‐dependent fatty acid metabolism controls the immune suppressive phenotype of tumor‐associated macrophages | EMBO Molecular Medicine
Metabolic reprogramming in the immunosuppression of tumor-associated macrophages | Chinese Medical Journal
Etomoxir Inhibits Macrophage Polarization by Disrupting CoA Homeostasis - ScienceDirect
Inhibition of Fatty Acid Oxidation Promotes Macrophage Control of Mycobacterium tuberculosis | mBio
Mitochondrial Metabolic Reprogramming by CD36 Signaling Drives Macrophage Inflammatory Responses | Circulation Research
Targeting the Key Enzymes of Abnormal Fatty Acid β-oxidation as a Potential Strategy for Tumor Therapy
Macrophage fatty acid oxidation inhibits atherosclerosis progression - Journal of Molecular and Cellular Cardiology
Frontiers | Macrophage Metabolism As Therapeutic Target for Cancer, Atherosclerosis, and Obesity